The hallmark of laryngeal obstruction is stridor. Inspiratory stridor indicates glottic or supraglottic obstruction, expiratory stridor bronchial obstruction and two-way stridor subglottic obstruction. If stridor is accompanied by cyanosis, tachycardia and intercostal and sternal recession, urgent measures are needed to save life. In less severe cases a hoarse voice, feeding problems and recurrent chest infections may occur.
Congenital laryngeal obstruction (先天性喉阻塞)
Aetiology:
Congenital anomalies include laryngeal cysts, webs, stenosis, vascular rings and vocal cord paralysis. Laryngomalacia is a condition caused by abnormal flaccidity of the larynx allowing the supraglottic structures to be drawn into the airway on inspiration; the condition resolves with age.
Management:
All cases of congenital stridor should undergo direct laryngoscopy.
Acquired laryngeal obstruction (後天性喉阻塞)
Aetiology:
- Acute epiglottitis – due to Haemophilus influenzae. It causes rapidly progressive airway obstruction.
- Acute laryngotracheobronchitis (croup) – due to para-influenzae virus or respiratory syncytial virus. It produces oedema, exudates and crusting of the larynx, trachea and bronchi.
- Subglottic stenosis – may follow infant tracheostomy or prolonged endotracheal intubation.
Management:
- Acute epiglottitis – treatment is with intravenous chloramphenicol. Endotracheal intubation or an emergency tracheostomy may be necessary.
- Acute laryngotracheobronchitis – endotracheal intubation is rarely required.
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